Neurology

Switching to fingolimod: TRANSFORMS extension study results

 

REPORT FROM THE 26TH CONGRESS OF THE EUROPEAN COMMITTEE FOR TREATMENT AND RESEARCH  IN MS (ECTRIMS), GOTHENBURG, SWEDEN, OCTOBER 13-16, 2010 – TRANSFORMS (Trials assessing injectable interferon vs. fingolimod oral in RRMS) was a phase III trial comparing fingolimod and intramuscular beta-interferon-1a in patients with relapsing-remitting MS (Cohen et al. N Engl J Med 2010; 362: 402-415; updated results in Clinical Developments in Multiple Sclerosis , Neurosens, July 7, 2010). The 12-month study reported annualized relapse rates (ARR) of 0.16 with fingolimod 0.5 mg/day, 0.20 with fingolimod 1.25 mg/day and 0.33 with beta-interferon-1a.

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TEMSO phase III results for teriflunomide

 

REPORT FROM THE 26TH CONGRESS OF THE EUROPEAN COMMITTEE FOR TREATMENT AND RESEARCH  IN MS (ECTRIMS), GOTHENBURG, SWEDEN, OCTOBER 13-16, 2010 – The novel oral agent teriflunomide reduces relapse rates by about one-third compared to placebo in patients with relapsing-remitting MS, according to new results from the TEMSO phase III trial presented at ECTRIMS (O’Connor et al. ECTRIMS 2010; abstract 79). The annualized relapse rate (ARR) was significantly reduced in both active treatment arms versus placebo: ARR was 0.370 and 0.369 with teriflunomide 7 mg/day and 14 mg/day, respectively, compared to 0.539 with placebo. The relative risk reductions with teriflunomide were 31.2% and 31.5%, respectively, with the two doses.

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Investigating interaction of environmental, genetic factors in MS

 

REPORT FROM THE 26TH CONGRESS OF THE EUROPEAN COMMITTEE FOR TREATMENT AND RESEARCH  IN MS (ECTRIMS), GOTHENBURG, SWEDEN, OCTOBER 13-16, 2010 – Recent papers have speculated that environmental factors, such as vitamin D status, may interact directly with genetic factors to increase the susceptibility to MS (Handunnetthi et al. Neurology 2010; 74: 1905-1910). Of particular interest is the HLA DRB1*1501 allele, which has been shown in numerous studies to influence MS risk (Link et al. J Neuroimmunol 2010; 226: 172-176).

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Benign MS: database analysis

 

REPORT FROM THE 26TH CONGRESS OF THE EUROPEAN COMMITTEE FOR TREATMENT AND RESEARCH  IN MS (ECTRIMS), GOTHENBURG, SWEDEN, OCTOBER 13-16, 2010 – An analysis of data from a longitudinal cohort of MS patients diagnosed in Gothenburg, Sweden (n=307), in the period 1950-1964 reports that 18% have remained non-progressive 45 years after disease onset (Andersen O. ECTRIMS 2010; abstract 36).

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CCSVI: interventions not justified

 

REPORT FROM THE 26TH CONGRESS OF THE EUROPEAN COMMITTEE FOR TREATMENT AND RESEARCH  IN MS (ECTRIMS), GOTHENBURG, SWEDEN, OCTOBER 13-16, 2010 – “We should not be using any interventions to treat CCSVI (chronic cerebrospinal venous insufficiency) until we can demonstrate that it has a pathological role,” stated Dr. Giancarlo Comi, Milan, Italy, at a special symposium organized by the European Charcot Foundation. “CCSVI is not a cause of MS,” he said, “and what is the evidence that it influences the MS disease process?”

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